[1] |
McKee AC, Stern RA, Nowinski CJ, et al. The spectrum of disease in chronic traumatic encephalopathy[J]. Brain, 2013, 136(Pt 1): 43-64.
|
[2] |
Mez J, Daneshvar DH, Kiernan PT, et al. Clinicopathological evaluation of chronic traumatic encephalopathy in players of American football[J]. JAMA, 2017, 318(4): 360-370.
|
[3] |
Arman FZ. Chronic traumatic encephalopathy: a brief overview[J]. Front Neurol, 2019, 10: 713.
|
[4] |
McKee AC, Stein TD, Kiernan PT, et al. The neuropathology of chronic traumatic encephalopathy[J]. Brain Pathol, 2015, 25(3): 350-364.
|
[5] |
Jordan BD. The clinical spectrum of sport-related traumatic brain injury[J]. Nat Rev Neurol, 2013, 9(4): 222-230.
|
[6] |
Stern RA, Daneshvar DH, Baugh CM, et al. Clinical presentation of chronic traumatic encephalopathy[J]. Neurology, 2013, 81(13): 1122-1129.
|
[7] |
Smith DH, Victoria EJ, John Q, et al. Chronic traumatic encephalopathy-confusion and controversies[J]. Nat Rev Neurol, 2019, 15(3): 179-183.
|
[8] |
Mawdsley C, Ferguson FR. Neurological disease in boxers[J]. Lancet, 1963, 2(7312): 795-801.
|
[9] |
Brandenburg W, Hallervorden J. Dementia pugilistica with anatomical findings[J]. Virchows Arch Pathol Anat Physiol Klin Med, 1954, 325(6): 680-709.
|
[10] |
Corsellis JA, Bruton CJ, Freeman-Browne D. The aftermath of boxing[J]. Psychol Med, 1973, 3(3): 270-303.
|
[11] |
McKee AC, Cairns NJ, Dickson DW, et al. The first NINDS/NIBIB consensus meeting to define neuropathological criteria for the diagnosis of chronic traumatic encephalopathy[J]. Acta Neuropathol, 2016, 131(1): 75-86.
|
[12] |
Omalu BI, DeKosky ST, Minster RL, et al. Chronic traumatic encephalopathy in a National Football League player[J]. Neurosurgery, 2005, 57(1): 128-134; discussion 128-134.
|
[13] |
Ropper AH. Links in the chain of chronic traumatic encephalopathy[J]. N Engl J Med, 2019, 380(18): 1771-1772.
|
[14] |
Stewart W, McNamara PH, Lawlor B, et al. Chronic traumatic encephalopathy: a potential late and under recognized consequence of rugby union?[J]. QJM, 2016, 109(1): 11-15.
|
[15] |
Ling H, Morris HR, Neal JW, et al. Mixed pathologies including chronic traumatic encephalopathy account for dementia in retired association football (soccer) players[J]. Acta Neuropathol, 2017, 133(3): 337-352.
|
[16] |
McKee AC, Cantu RC, Nowinski CJ, et al. Chronic traumatic encephalopathy in athletes: progressive tauopathy after repetitive head injury[J]. J Neuropathol Exp Neurol, 2009, 68(7): 709-735.
|
[17] |
Falcon B, Zivanov J, Zhang W, et al. Novel tau filament fold in chronic traumatic encephalopathy encloses hydrophobic molecules[J]. Nature, 2019, 568(7752): 420-423.
|
[18] |
Stern RA, Adler CH, Chen K, et al. Tau positron-emission tomography in National Football League players[J]. N Engl J Med, 2019, 380(18): 1716-1725.
|
[19] |
Johnson VE, Stewart W, Smith DH. Traumatic brain injury and amyloid-β pathology: a link to Alzheimer’s disease?[J]. Nat Rev Neurosci, 2010, 11(5): 361-370.
|
[20] |
Roberts GW, Allsop D, Bruton C. The occult aftermath of boxing[J]. J Neurol Neurosurg Psychiatry, 1990, 53(5): 373-378.
|
[21] |
Stein TD, Montenigro PH, Alvarez VE, et al. Beta-amyloid deposition in chronic traumatic encephalopathy[J]. Acta Neuropathol, 2015, 130(1): 21-34.
|
[22] |
Josephs KA, Dickson DW, Tosakulwong N, et al. Rates of hippocampal atrophy and presence of post-mortem TDP-43 in patients with Alzheimer’s disease: a longitudinal retrospective study[J]. Lancet Neurol, 2017, 16(11): 917-924.
|
[23] |
Deng Z, Lim J, Wang Q, et al. ALS-FTLD-linked mutations of SQSTM1/p62 disrupt selective autophagy and NFE2L2/NRF2 anti-oxidative stress pathway[J]. Autophagy, 2020, 16(5): 917-931.
|
[24] |
McKee AC, Gavett BE, Stern RA, et al. TDP-43 proteinopathy and motor neuron disease in chronic traumatic encephalopathy[J]. J Neuropathol Exp Neurol, 2010, 69(9): 918-929.
|
[25] |
Saing T, Dick M, Nelson PT, et al. Frontal cortex neuropathology in dementia pugilistica[J]. J Neurotrauma, 2012, 29(6): 1054-1070.
|
[26] |
Sidaros A, Skimminge A, Liptrot MG, et al. Long-term global and regional brain volume changes following severe traumatic brain injury: a longitudinal study with clinical correlates[J]. Neuroimage, 2009, 44(1): 1-8.
|
[27] |
Xu Y, McArthur DL, Alger JR, et al. Early nonischemic oxidative metabolic dysfunction leads to chronic brain atrophy in traumatic brain injury[J]. J Cereb Blood Flow Metab, 2010, 30(4): 883-894.
|
[28] |
Johnson VE, Stewart W, Smith DH. Widespread τ and amyloid-β pathology many years after a single traumatic brain injury in humans[J]. Brain Pathol, 2012, 22(2): 142-149.
|
[29] |
Chen XH, Siman R, Iwata A, et al. Long-term accumulation of amyloid-beta, beta-secretase, presenilin-1, and caspase-3 in damaged axons following brain trauma[J]. Am J Pathol, 2004, 165(2): 357-371.
|
[30] |
Ikonomovic MD, Uryu K, Abrahamson EE, et al. Alzheimer’s pathology in human temporal cortex surgically excised after severe brain injury[J]. Exp Neurol, 2004, 190(1): 192-203.
|
[31] |
Smith DH, Stewart W. Traumatic brain injury: a platform for studies in Aβ processing: commentary on: "Rapid Aβ oligomer and protofibril accumulation in traumatic brain injury" [J]. Brain Pathol, 2018, 28(4): 463-465.
|
[32] |
Wang HK, Lee YC, Huang CY, et al. Traumatic brain injury causes frontotemporal dementia and TDP-43 proteolysis[J]. Neuroscience, 2015, 300: 94-103.
|